Essays on Implications of Type 1 Diabetes-Long Term Condition on a Patients Life Journey and Wellbeing Case Study

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The paper “ Implications of Type 1 Diabetes-Long Term Condition on a Patient’ s Life Journey and Wellbeing” is a forceful example of a finance & accounting case study. Type 1 diabetes constitutes 5-15% of 285 million adults with diabetes. The disease is increasing at 3% annually, particularly in white Europeans, including northern Europe (Hovorka et al 2011). It accounts for almost 57000 death every year among under 75years, the most intensive users of high-cost services (DoH, 2008). As one of the diseases associated with long-term conditions, type 1 diabetes is a global concern.

Approximately 17.5 million people have a long-term condition (Margereson and Trenoweth, 2010). In Britain, six out of ten adults have a long-term condition that can’ t be cured thus making their care and management even more complex (Chronic disease management, 2004). The aim of this paper is to present a detailed discussion on the implications of a Type 1 diabetes long-term condition on a patient’ s life journey and wellbeing with the use of a case study of a patient suffering from the disease. Background to LTC-Type 1 diabetesPeople with Type 1 diabetes are generally are not obese and may present initially with diabetic ketoacidosis (DKA).

The distinguishing characteristic of a patient with type 1 diabetes is that if his or her insulin is withdrawn, ketosis and eventually ketoacidosis develop. Therefore, these patients are dependent on exogenous insulin which calls for a multidisciplinary approach by physician, nurse, and dietitian. At the new onset of type 1 diabetes, permanent insulin therapy has to be started. There is also need for a long-term medical attention both to limit the development of its devastating complications and to manage them when they do occur.

Type 1 diabetes is an autoimmune disease that results when the body’ s system for fighting infection— the immune system— turns against a part of the body. In this case, the immune system attacks and destroys the insulin-producing beta cells in the pancreas (Devendra, 2004). It is related to premature beta-cell failure or pancreatic beta-cell destruction where the patients completely lack insulin secretion leading to increased blood and urine glucose or severe metabolic lability and eventually ketoacidosis.


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